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Chapman IM, Goble EA, Wittert GA, Morley JE & Horowitz M 1998 ; . Effect of intravenous glucose and euglycemic insulin infusions on short-term appetite and food intake. J Physiol 274, R596R603. Chen HY, Trumbauer ME, Chen AS, Weingarth DT, Adams JR, Frazier EG, et al. Nargund RP, Smith RG, Van Der Ploeg LH, Howard AD, MacNeil DJ & Qian S 2004 ; . Orexigenic action of peripheral ghrelin is mediated by neuropeptide Y NPY ; and agouti-related protein AgRP ; . Endocrinology 145, 26072612. Clapham JC, Arch JR & Tadayyon M 2001 ; . Anti-obesity drugs: a critical review of current therapies and future opportunities. Pharmacol Therap 89, 81121. Cohen MA, Ellis SM, Le Roux CW, Batterham RL, Park A, Patterson M et al. 2003 ; . Oxyntomodulin suppresses appetite and reduces food intake in humans. J Clin Endocrinol Metab 88, 46964701. Considine RV, Sinha MK, Heiman ML, Kriauciunas A, Stephens TW, Nyce MR et al. 1996 ; . Serum immunoreactive-leptin concentrations in normal-weight and obese humans. N Engl J Med 334, 292295. Cowley MA, Smith RG, Diano S, Tschop M, Pronchuk N, Grove KL et al. 2003 ; . The distribution and mechanism of action of ghrelin in the CNS demonstrates a novel hypothalamic circuit regulating energy homeostasis. Neuron 37, 649661. Crawley JN & Beinfeld MC 1983 ; . Rapid development of tolerance to the behavioural actions of cholecystokinin. Nature 302, 703706. Cummings DE, Purnell JQ, Frayo RS, Schmidova K, Wisse BE & Weigle DS 2001 ; . A preprandial rise in plasma ghrelin levels suggests a role in meal initiation in humans. Diabetes 50, 17141719. Cummings DE, Weigle DS, Frayo RS, Breen PA, Ma MK et al. 2002 ; . Plasma ghrelin levels after diet-induced weight loss or gastric bypass surgery. N Engl J Med 346, 16231630. Dakin CL, Small CJ, Batterham RL, Neary NM, Cohen MA, Patterson M et al. 2004 ; . Peripheral oxyntomodulin reduces food intake and body weight gain in rats. Endocrinology 145, 26872695. Dakin CL, Small CJ, Park AJ, Seth A, Ghatei MA & Bloom SR 2002 ; . Repeated ICV administration of oxyntomodulin causes a greater reduction in body weight gain than in pair-fed rats. J Physiol 283, E1173E1177. Date Y, Kojima M, Hosoda H, Sawaguchi A, Mondal MS, Suganuma T et al. 2000 ; . Ghrelin, a novel growth hormone-releasing acylated peptide, is synthesized in a distinct endocrine cell type in the gastrointestinal tracts of rats and humans. Endocrinology 141, 42554261. Ellacott KL & Cone RD 2004 ; . The central melanocortin system and the integration of short- and long-term regulators of energy homeostasis. Recent Prog Horm Res 59, 395408, for instance, side affects.
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Mennella, J.A., and Gerrish, C.J. Effects of exposure to alcohol in mothers' milk on the infants' sleep and activity levels. Pediatrics, in press. 19 ; Block, A.J., et al. Effect of alcohol ingestion on breathing and oxygenation during sleep. J Med 80 4 ; : 595-600, 1986. 20 ; Allen, R.P., et al. Electroencephalographic EEG ; sleep recovery following prolonged alcohol intoxication in alcoholics. J Ner and Ment Dis 153 6 ; : 424433, 1971. 21 ; Williams, H.L., and Rundell, Jr., O.H. Altered sleep physiology in chronic alcoholics: Reversal with abstinence. Alcohol Clin Exp Res 5 2 ; : 318-325, 1981. 22 ; Gillin, J.C., et al. EEG sleep studies in "pure" primary alcoholism during subacute withdrawal: Relationships to normal controls, age, and other clinical variables. Bio Psychiatry 27: 477-488, 1990. ; Lester, B.K., et al. Chronic alcoholism, alcohol and sleep. In: Gross, M.M., ed. Advances in Experimental Medicine and Biology: Volume 35. Alcohol Intoxication and Withdrawal: Experimental Studies. New York: Plenum Press, 1973. pp. 261-279. 24 ; Skoloda, T.E., et al. Sleep quality reported by drinking and non-drinking alcoholics. In: Gottheil, E.L., et al., eds. Addiction Research and Treatments: Converging Trends. New York: Pergamon Press, 1979. pp. 102-112. 25 ; Zarcone, V., et al. Alcohol, sleep and cerebrospinal fluid changes in alcoholics: Cyclic AMP and biogenic amine metabolites in CSF. In: Gross, M.M., ed. Advances in Experimental Medicine and Biology: Volume 85A. Alcohol Intoxication and Withdrawal--IIIa: Biological Aspects of Ethanol. New York: Plenum Press, 1977. pp. 593-599. 26 ; Gillin, J.C., et al. Increased pressure for rapid eye movement sleep at time of hospital admission predicts relapse in nondepressed patients with primary alcoholism at 3-month follow-up. Arch Gen Psychiatry 51: 189-197, 1994, for example, esidrix.
Acknowledgements: this paper has been supported by the medical university of lodz, research project no 503-102-4 "studies on the effects of neurohormones, their analogs and antagonists on proliferation and apoptosis in normal and neoplastic tissues and raloxifene.
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The possible protective effects against seizures provided by polyunsaturated fatty acids, especially fatty acids of the n-3 group EPA and DHA ; have been previously reported in the literature. Some additional support can be found in a recent review on the beneficial effects of a ketogenic diet.75 Similarly, the role of fatty acids in increasing the threshold for electrically induced seizures has been established in a series of meticulously executed in vitro research studies.76 Confirmatory in vivo research using the mouse has shown that n-3 fatty acids are able to block PTZ induced unhealthy brain excitation and stimulation that produces convulsions in various hippocampal neurons.49 While it seems that essential fatty acids are neuroprotective, the relative potency of the separate FAs is not known. From our laboratory findings, and from the work of others, 19 we believe that a ratio of 1: 4 alpha-linolenic ALA ; to linoleic acid LA ; is the optimal neuroprotective combination. Some investigators, however, have proposed that even with an adequate LA and ALA levels, seizure patients may lack sufficient enzymatic activity necessary to metabolize these FAs, and that supplementation with EPA and or DHA could be more attractive clinical design for near term clinical experimentation. We believe that even without waiting for the results of formal designs of randomized clinical trials the safety of dietary PUFA supplementation when taken in the recommended dosages ; is significant to warrant implementation at this time, on a case by case basis.
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1. Ward MM, Javitz HS, Smith WM, Bakst A. Direct medical cost of chronic obstructive pulmonary disease in the U.S.A. Respir Med 2000; 94 11 ; : 11231129. 2. Halpern MT, Stanford RH, Borker R. The burden of COPD in the U.S.A.: Results from the Confronting COPD Survey. Respir Med 2003; 97 Suppl 3 ; : S81S89. 3. Vermeire P. The burden of chronic obstructive pulmonary disease. Respir Med 2002; 96 Suppl C ; : S3S10. 4. Carter R, Blevins W, Stocks J, et al. Cost and quality issues related to the management of COPD. Semin Respir Crit Care Med 1999; 20 3 ; : 199212. 5. Division of Epidemiology, National Heart, Lung, and Blood Institute, National Institutes of Health. Data Fact Sheet 2003. Available at: nhlbi. nih.gov health public lung other copd fact . Accessed April 2006. 6. Mapel D, Hurley J, Frost F, et al. Health care utilization in chronic obstructive pulmonary disease: A case-control study in a health maintenance organization. Arch Intern Med 2000; 160 17 ; : 2653 2658. 7. Stewart AL, Greenfield S, Hays RD, et al. Functional status and wellbeing of patients with chronic conditions: Results from the Medical Outcomes Study. JAMA 1989; 262: 907913. Van Der Molen T, Pieters W, Bellamy D, Taylor R. Measuring the success of treatment for chronic obstructive pulmonary disease patient, physician and health care payer perspective. Respir Med 2002; 96 Suppl C ; : S17S21 and vaseretic and esidrix, for example, fda.
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